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| Product: |
Nutrition Supplies Practitioner Range GABA 600g |
| Code: |
9338567010417 |
| RRP: |
AUD 150.00 |
| Was: |
AUD 100.00 |
| Now: |
AUD 79.00 |
| Availability: |
IN STOCK
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| Description: |
Nutrition Supplies GABA - Growth Hormone Release 100% Pure GABA
What is Gamma Aminobutyric Acid (GABA)?
GABA (gamma-aminobutric acid) is the most important and abundant inhibitory neurotransmitter in the brain (it's actually an amino acid classified as a neurotransmitter). It helps induce relaxation and sleep. It acts as a "balancer" for the brain where excitation of the brain is balanced with inhibition.
GABA and HGH:
GABA stimulates the anterior pituitary, leading to higher levels of Human Growth Hormone (HGH), and if you didn't know, HGH is very anabolic (builds muscle) and lipotropic (fat burning). So GABA indirectly has anti-ageing effects as well.
GABA Research:
There have been many studies done on GABA showing its effect on increased HGH.
Cavagnini, F., et al. Effect of acute and repeated administration of gamma aminobutyric acid (GABA) on growth hormone and prolactin secretion in man. Acta Endocrinol (Copenhagen). 93(2):149-154, 1980.
This study found that a single dose of 5,000 mg of GABA administered to 19 subjects significantly elevated plasma hGH levels compared to placebo-treated control subjects. There was a 5½ fold increase in hGH levels 90 minutes after GABA administration.
Warning: GABA also inhibits HGH if consumed while exercising.
Coiro, V., et al. Opioid modulation of the gamma-aminobutyric acid-controlled inhibition of exercise-stimulated growth hormone and prolactin secretion in normal men. Eur J Endocrinol. 131(1):50-55, 1994.
Department of Internal Medicine, School of Medicine, University of Parma, Italy.
The possible involvement of endogenous opioids in the gamma-aminobutyric acid-controlled (GABAergic) inhibition of growth hormone (GH) and prolactin (PRL) during physical exercise was evaluated in normal men. After fasting overnight, seven subjects were tested on four mornings at least 1 week apart. Exercise was performed on a bicycle ergometer. The workload was gradually increased at 3-min intervals until exhaustion and lasted about 15 min in all subjects. Tests were carried out under administration of placebo, the opioid antagonist naloxone (10 mg as an iv bolus injection), the GABAergic agonist sodium valproate (600 mg in three divided doses orally) or naloxone plus sodium valproate. During exercise, plasma GH and PRL levels rose 5.5
- and 1.9-fold, respectively. The administration of naloxone did not modify, whereas sodium valproate significantly reduced the plasma GH and PRL rise during exercise. In the presence of sodium valproate, GH and PRL levels rose 3- and 1.5-fold, respectively, in response to exercise. When naloxone was given together with sodium valproate, both GH and PRL responses to exercise were abolished completely. These data suggest the involvement of a GABAergic mechanism in the regulation of GH and PRL responses to physical exercise in men. Furthermore, the data argue against a role of naloxone-sensitive endogenous opioids in the control of these hormonal responses to exercise, whereas they suggest a modulation by opioids of the GABAergic inhibitory action.
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